Tiv tauj peb
- Chav 1204,|| Vanke Huizhi Center, Yog Koog tsev kawm ntawv, Xi'an Lub zos, Shaanxi, Tuam Tshoj
- Chriswang@sheerherb.com
- +86 132 8987 3310
Benfotiamine hmoov
Khoom npe Benfotiaminec
Lub npe Latin Benfotiamine
Tshooj Siv S-benzoylthiamine O-monophosphate
Qhov tshwm sim Dawb los yog tawm-dawb crystalline hmoov
Cov khoom xyaw nquag Benfotiamine
Purity 99 feem pua
CAS Nr. 22457-89-2
cia txias qhov chaw qhuav
qib zaub mov qib
Hauj lwm lawm
Benfotiamine hmoov

KhoomTuag
Dab tsi yogBenfotiamine Hmoov?
Nws yog ib tug fat-soluble analogue ntawm vitamin B1, muaj ib tug ntau dua absorbing tus nqi thiab bioavailability tshaj vitamin B1, thiab feem ntau yog siv los ua zaub mov ntxiv rau cov mob ntshav qab zib. Benfotiamine muaj peev xwm tiv thaiv oxidant ncaj qha los tiv thaiv DNA puas.
Benfotiamineyog vitamin B1 rog soluble derivatives, txhim kho lub bioavailability ntawm dej-soluble vitamin B1 tsawg shortcomings, txhim kho cov ntshav thiab cov ntaub so ntswg concentrations ntawm thiamine, thiaj li mus txhim kho cov nyhuv curative.
Benfotiamine feem ntau yog ua lag luam raws li kev noj haus antioxidant ntxiv. Hauv kev tshawb fawb soj ntsuam nrog rau rau tus neeg mob, benfotiamine txo qis AGE los ntawm 40 feem pua.

| Cov ntaub ntawv yooj yim | |
| Khoom npe | Benfotiamine |
| Lub npe Latin | Benfotiamine |
| Ib feem siv | S- benzoylthiamineO- monophosphate |
| Qhov tshwm sim | Dawb los yog dawb crystalline hmoov |
| Cov khoom xyaw nquag | Benfotiamine |
| Purity | 99 feem pua |
| CAS Nr. | 22457-89-2 |
| khaws cia | txias qhov chaw qhuav |
| qib | qib zaub mov |
Khoom Specifications
Yam khoom | Specifications | Cov txiaj ntsig |
Qhov tshwm sim | Dawb los yog yuav luag dawb crystalline hmoov | Ua raws |
Kev txheeb xyuas | ||
Infrared nqus | Raws li kev siv IR spectra | Ua raws |
Thiamine | Tsawg dua lossis sib npaug rau 0.1 feem pua | 0.05 feem pua |
pH | 2.7~3.3 | 2.9 |
Dej | Tsawg dua lossis sib npaug rau 1.0 feem pua | 0.15 feem pua |
Residue ntawm ignition | Tsawg dua lossis sib npaug rau 0.1 feem pua | 0.02 feem pua |
Cov khoom muaj feem xyuam | ||
impurity A | Tsawg dua lossis sib npaug rau 0.15 feem pua | Ua raws |
Impurity B | Tsawg dua lossis sib npaug rau 0.3 feem pua | Ua raws |
Cov impurity C | Tsawg dua lossis sib npaug rau 0.15 feem pua | Ua raws |
Unspecified impurity | Tsawg dua lossis sib npaug rau 0.10 feem pua | Ua raws |
Tag nrho impurities | Tsawg dua lossis sib npaug rau 0.5 feem pua | Ua raws |
Cov tshuaj sib txuas | Tsawg dua lossis sib npaug rau 1.0 feem pua | Ua raws |
Cov hlau hnyav | Tsawg dua lossis sib npaug li 20ppm | Ua raws |
Assay | 98.0~102.0 feem pua | 99.6% ua |
Khoom muaj nuj nqi
1. Mob ntshav qab zib neuropathy
BenfotiamineKev kho mob zoo li yog ib lub tswv yim zoo rau kev tiv thaiv kab mob ntshav qab zib nephropathy, ib qho teeb meem loj ntawm ntshav qab zib uas cuam tshuam nrog kev pheej hmoo siab ntawm kab mob plawv thiab kev tuag. Cov piam thaj ntau ntau ua rau cov khoom txuam ntawm triose phosphate; Qhov no ua rau biochemical tsis ua haujlwm thiab qhov kawg ntawm kev txhim kho ntshav qab zib nephropathy.
Cov kev tshawb fawb preclinical hauv nas pom tias benfotiamine tiv thaiv kev sib sau ntawm triose phosphate hauv ntshav qab zib, yog li inhibiting kev loj hlob ntawm cov kab mob raum.
2. Kev paub txog kev txhim kho hauv Alzheimer's disease
PreclinicalCov kev tshawb fawb qhia tias benfotiamine muaj txiaj ntsig zoo rau kev paub tsis meej hauv Alzheimer's disease. Benfotiamine tau pom tias txo cov amyloid deposits hauv tsiaj cov qauv ntawm Alzheimer's kab mob. Txawm li cas los xij, cov teebmeem no tsis tau tshwm sim hauv tib neeg. Ib theem II kev sim tshuaj ntsuam xyuas tam sim no ntsuas seb benfotiamine tuaj yeem tiv thaiv kev paub tsis zoo ntawm cov neeg uas muaj kev paub tsis meej me ntsis.
3. kov yeej kev haus cawv
Cawv cawvKev noj haus yog txuam nrog qhov tsis txaus ntawm cov as-ham tseem ceeb thiab cov vitamins. Alcoholism-related thiamine deficiency yog tus cwj pwm los ntawm cov kab mob neurological vim qhov poob ntawm qhov nruab nrab thiab qhov nruab nrab ntawm cov khoom dawb. Txawm hais tias kev hloov thiamine yog qhov tseem ceeb hauv cov neeg mob uas muaj cawv vim raug mob CNS, benfotiamine tsis tau pom tias muaj txiaj ntsig zoo hauv peripheral nkag mus rau CNS.
4. Benfotiamine rau kev haus luam yeeb
Kev haus luam yeebua rau endothelial dysfunction, ib qho tshwm sim thaum ntxov hauv kab mob plawv. Oxidative stress thiab o tuaj yeem ua rau qhov no ntau dua. Hauv kev tshawb fawb ua ntej hauv nas, benfotiamine tau pom los tiv thaiv nicotine-induced vascular dysfunction. Ib qho kev sim hauv cov neeg tuaj yeem noj qab haus huv tau pom tias kev kho mob luv luv nrog benfotiamine ib nrab rov ua haujlwm macrovascular hauv cov neeg haus luam yeeb noj qab haus huv.
5. Kev tiv thaiv raum
Cov ntaub ntawv los ntawmCov kev tshawb fawb nas qhia tias benfotiamine muaj kev tiv thaiv ntawm peritoneum thiab lub raum seem thaum lub sij hawm peritoneal dialysis thiab uremia. Raws li kev tshawb fawb luam tawm nyob rau hauv Phau Ntawv Xov Xwm ntawm American Academy of Nephrology, ntxiv benfotiamine rau kev noj haus ua rau muaj kev ua haujlwm ntawm transketolase thiab txo qis kev qhia txog qib siab glycation kawg cov khoom thiab lawv cov receptors.
Kev tshawb fawb los ntawm Ustuner et al. Kev tshawb fawb luam tawm nyob rau hauv cov ntaub so ntswg thiab Cell pom tias benfotiamine supplementation yuav pab txo tau cov tshuaj tua kab mob-induced nephrotoxicity los ntawm kev txhim kho oxidative kev nyuaj siab thiab o nyob rau hauv lub raum tubular hlwb.
6. Muaj peev xwm tiv thaiv qog
Cov kev tshawb fawb hauv vitro qhia tiastshuaj benfotiaminetuaj yeem thaiv cov kab mob leukemia los ntawm lub tshuab hu ua apoptosis. Paposis yog ib hom kev tuag ntawm tes nrog kev tsim ntawm cytoplasmic vacuoles. Kev tshawb fawb txog kab lis kev cai ntawm tes los ntawm cov kws tshawb fawb ntawm Kanazawa University hauv Nyij Pooj tau pom tias benfotiamine yog cov tshuaj muaj txiaj ntsig zoo rau cov neeg mob uas mob ntshav qab zib mellitus uas tsis tsim nyog rau kev kho tshuaj khomob. Lawv kuj tau pom tias benfotiamine tau ua haujlwm sib koom ua ke nrog cov tshuaj khomob cytarabine txhawm rau tshem tawm cov cell myeloid leukemia.
7. Antioxidant zog
BenfotiamineNws kuj yog ib qho muaj zog antioxidant nrog cov tshuaj tiv thaiv kab mob los ntawm inhibiting synthesis ntawm prostaglandins thiab leukotrienes.
Daim ntawv thov teb Benfotiamine hmoov
1. Benfotiamine yog siv los tiv thaiv thiab kho cov vitamin B1 tsis txaus;
2. Rau vitamin B1 xav tau nce, nrog kev tsis txaus los ntawm cov khoom noj khoom haus (kev qaug zog, hyperthyroidism, cev xeeb tub, lactation, thaum lub sijhawm ua haujlwm hnyav, thiab lwm yam);
3. Benfotiamine yog siv rau hauv kev kho mob uas tsis yog-alcoholic wernicke encephalopathy;
4. Kho beriberi;
5. Siv nyob rau hauv cov kab mob hauv qab no, speculation thiab vitamin B1 deficiency thiab metabolic mob cuam tshuam nrog kev kho mob, xws li: neuralgia; mob nqaij, mob pob qij txha; Peripheral neuritis, peripheral paj palsy; Myocardial metabolic ntshawv siab; Kev cem quav thiab lwm yam kab mob plab hnyuv ua haujlwm cuam tshuam
6. Rau kev kho mob Alzheimer's adjuvant
7. Qhov cuam tshuam ntawm kev kho nicotine rau vascular dysfunction nyob rau hauv cov neeg haus luam yeeb
8. Antioxidant thiab anti-inflammatory

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